Part of the physiological process of erection involves the release of nitric oxide (NO) in the corpus cavernosum. This then activates the enzyme guanylate cyclase which results in increased levels of cyclic guanosine monophosphate (cGMP), leading to smooth muscle relaxation in the corpus cavernosum, resulting in increased inflow of blood and an erection. Sildenafil is a potent and selective inhibitor of cGMP specific phosphodiesterase type 5 (PDE5) which is responsible for degradation of cGMP in the corpus cavernosum. This means that, ...

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Sildenafil, Sildenafil - History, Sildenafil - Mechanism of action, Sildenafil - Dosage and price, Sildenafil - Contraindications and side effects, Sildenafil - Uses outside of erectile dysfunction, Sildenafil - Pulmonary hypertension, Sildenafil - Raynaud's phenomenon

Read more here: » Sildenafil: Encyclopedia II - Sildenafil - Mechanism of action

Local anesthetic drugs act mainly by inhibiting sodium influx through sodium-specific ion channels in the neuronal cell membrane, in particular the so-called voltage-gated sodium channels. When the influx of sodium is interrupted, an action potential cannot arise and signal conduction is thus inhibited. The receptor site is thought to be located at the cytoplasmic (inner) portion of the sodium channel. Local anesthetic drugs selectively bind to activated and inactivated sodium channel (without binding to sodium channel in resting membrane ...

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Local anesthetic, Local anesthetic - Mechanism of action, Local anesthetic - Undesired effects, Local anesthetic - Central nervous system, Local anesthetic - Cardiovascular system, Local anesthetic - Hypersensitivity/Allergy, Local anesthetic - Methemoglobinemia, Local anesthetic - Clinically used local anesthetics

Read more here: » Local anesthetic: Encyclopedia II - Local anesthetic - Mechanism of action

The primary mechanism of action responsible for Ketorolac's anti-inflammatory/antipyretic/analgesic effects is the inhibition of prostaglandin synthesis by competitive blocking of the the enzyme cyclooxygenase (COX). Like most NSAIDs, Ketorolac is a non-selective cyclooxygenase inhibitor. As with other NSAIDs, the mechanism of the drug is associated with the chiral S form. Conversion of the R enantiomer into the S enantiomer has been shown to occur in the metabolism of ibuprofen; it is unknown ...

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Ketorolac, Ketorolac - Chemistry, Ketorolac - Mechanism of action, Ketorolac - Indications, Ketorolac - Contraindications, Ketorolac - Adverse effects, Ketorolac - Warnings and precautions, Ketorolac - Notes, Ketorolac - Dosage availability and price

Read more here: » Ketorolac: Encyclopedia II - Ketorolac - Mechanism of action

Ibuprofen is an NSAID which is believed to work through inhibition of cyclooxygenase (COX); thus inhibiting prostaglandin synthesis. As with other NSAIDs, ibuprofen inhibits platelet aggregation, but is not used therapeutically for this action since it is a minor and reversible effect. Main article: Non-steroidal anti-inflammatory drug ...

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Ibuprofen, Ibuprofen - Clinical use, Ibuprofen - Indications, Ibuprofen - Off-Label and investigational use, Ibuprofen - Ibuprofen lysine, Ibuprofen - Mechanism of action, Ibuprofen - Adverse effects, Ibuprofen - Reported ADRs, Ibuprofen - Photosensitivity, Ibuprofen - Cardiovascular risk, Ibuprofen - Stereochemistry, Ibuprofen - Human toxicology, Ibuprofen - Availability

Read more here: » Ibuprofen: Encyclopedia II - Ibuprofen - Mechanism of action

Indomethacin is a nonselective inhibitor of cyclooxygenase (COX) 1 and 2, enzymes that participate in prostaglandin synthesis from arachidonic acid. Prostaglandins are hormone-like molecules normally found in the body, where they have a wide variety of effects, some of which lead to pain, fever, and inflammation. Prostaglandins also cause uterine contractions in pregnant women. Indomethacin is an effective tocolytic agent, able to delay premature labor by reducing uterine contractions through inhibition of PG synthesis in the uterus a ...

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Indomethacin, Indomethacin - Chemical properties, Indomethacin - Indications, Indomethacin - Contraindications, Indomethacin - Mechanism of action, Indomethacin - Adverse effects, Indomethacin - Necessary Examinations during Longterm Treatment, Indomethacin - Animal Toxicity and Human Overdose, Indomethacin - Usual Dosage Forms, Indomethacin - History

Read more here: » Indomethacin: Encyclopedia II - Indomethacin - Mechanism of action

Vancomycin acts by inhibiting proper cell wall synthesis in Gram-positive bacteria. The mechanism inhibited, and various factors related to entering the outer membrane of Gram-negative organisms mean that vancomycin is not active against Gram-negative bacteria. Specifically, vancomycin prevents incorporation of N-acetylmuramic acid (NAM)- and N-acetylglucosamine (NAG)-peptide subunits from being incorporated into the peptidoglycan matrix; which forms the major ...

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Vancomycin, Vancomycin - Mechanism of action, Vancomycin - Therapeutic considerations, Vancomycin - Clinical indications, Vancomycin - Resistance

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