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Mechanism of action | A Wisdom Archive on Mechanism of action | | Mechanism of action A selection of articles related to Mechanism of action | |
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Mechanism of action
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| ARTICLES RELATED TO Mechanism of action | | | | | |  Mechanism of action: Encyclopedia II - Ketorolac - Mechanism of actionThe primary mechanism of action responsible for Ketorolac's anti-inflammatory/antipyretic/analgesic effects is the inhibition of prostaglandin synthesis by competitive blocking of the the enzyme cyclooxygenase (COX). Like most NSAIDs, Ketorolac is a non-selective cyclooxygenase inhibitor.
As with other NSAIDs, the mechanism of the drug is associated with the chiral S form. Conversion of the R enantiomer into the S enantiomer has been shown to occur in the metabolism of ibuprofen; it is unknown ...
See also:Ketorolac, Ketorolac - Chemistry, Ketorolac - Mechanism of action, Ketorolac - Indications, Ketorolac - Contraindications, Ketorolac - Adverse effects, Ketorolac - Warnings and precautions, Ketorolac - Notes, Ketorolac - Dosage availability and price Read more here: » Ketorolac: Encyclopedia II - Ketorolac - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Paracetamol - Mechanism of action Paracetamol has long been suspected of having a similar mechanism of action to aspirin because of the similarity in structure. That is, it has been assumed that paracetamol acts by reducing production of prostaglandins, which are involved in the pain and fever processes, by inhibiting the cyclooxygenase (COX) enzyme.
However, there are important differences between the effects of aspirin and those of paracetamol. Prostaglandins participate in the inflammatory response, but paracetamol has no appreciable anti-inflammatory action. Furth ...
See also:Paracetamol, Paracetamol - History, Paracetamol - Available forms, Paracetamol - Mechanism of action, Paracetamol - Metabolism, Paracetamol - Toxicity, Paracetamol - Overview, Paracetamol - Mechanism of toxicity, Paracetamol - Risk factors for toxicity, Paracetamol - Natural history, Paracetamol - Diagnosis, Paracetamol - Treatment Read more here: » Paracetamol: Encyclopedia II - Paracetamol - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Neurotransmitter - Mechanism of action Within the cells, small-molecule neurotransmitter molecules are usually packaged in vesicles. When an action potential travels to the synapse, the rapid depolarization causes calcium ion channels to open. Calcium then stimulates the transport of vesicles to the synaptic membrane; the vesicle and cell membrane fuse, leading to the release of the packaged neurotransmitter, a mechanism called exocytosis.
The neurotransmitters then diffuse across the synaptic cleft to bind to receptors. The receptors are broadly classified into ionotropic ...
See also:Neurotransmitter, Neurotransmitter - Types of neurotransmitters, Neurotransmitter - Mechanism of action, Neurotransmitter - Post-synaptic effect, Neurotransmitter - Specific actions, Neurotransmitter - Common neurotransmitters, Neurotransmitter - Amino acids, Neurotransmitter - Biogenic amines, Neurotransmitter - Monoamines, Neurotransmitter - Polypeptides neuropeptides Read more here: » Neurotransmitter: Encyclopedia II - Neurotransmitter - Mechanism of action |
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| | | | | | Mechanism of action: Encyclopedia II - Diclofenac - Mechanism of action The exact mechanism of action is not entirely known, but it is thought that the primary mechanism responsible for its anti-inflammatory/antipyretic/analgesic action is inhibition of prostaglandin synthesis by inhibition of cyclooxygenase (COX).
Diclofenac, it seems, may also be a unique member of the NSAIDs. There is some evidence that diclofenac inhibits the lipooxygenase pathways, thus reducing formation of the leukotrienes (also pro-inflammatory autacoids). There is also speculation that diclofenac may inhibit phospholipase A2 ...
See also:Diclofenac, Diclofenac - Mechanism of action, Diclofenac - Common uses, Diclofenac - Off label/investigational uses, Diclofenac - Contraindications, Diclofenac - Side effects, Diclofenac - Veterinary information, Diclofenac - Interactions, Diclofenac - Dosage, Diclofenac - Animal toxicity and human overdose, Diclofenac - Environmental problems, Diclofenac - Reference Read more here: » Diclofenac: Encyclopedia II - Diclofenac - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Heparin - Mechanism of action Heparin works by potentiating the action of antithrombin III, as it is similar to the heparan sulfate proteoglycans which are naturally present on the cell membrane of the endothelium. Because antithrombin III inactivates many coagulation proteins, the process of coagulation will slow down.
The effects of heparin are measured in the lab by the partial thromboplastin time (aPTT), (the time it takes the blood plasma to clot).
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See also:Heparin, Heparin - History, Heparin - Mechanism of action, Heparin - Administration, Heparin - Medical use, Heparin - Other uses, Heparin - Treatment of overdose, Heparin - Adverse reactions Read more here: » Heparin: Encyclopedia II - Heparin - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Nadolol - Mechanism of action Nadolol non-selectively blocks beta-1 adrenergic receptors mainly in the heart, inhibiting the effects of the catecholamines epinephrine and norepinephrine and decreasing heart rate and blood pressure. It also blocks beta-2 adrenergic receptors located in bronchiole smooth muscle, causing vasoconstriction. By binding beta-2 receptors in the juxtaglomerular apparatus, nadolol inhibits the production of renin, thereby inhibiting angiotensin II and aldosterone production. Nadolol therefore inhibits the vasoconstriction and water retention due to angiotensin II and aldosterone, respectively.
The drug impai ...
See also:Nadolol, Nadolol - Chemistry and pharmacokinetics, Nadolol - Mechanism of action, Nadolol - Indications, Nadolol - Contraindications, Nadolol - Side effects Read more here: » Nadolol: Encyclopedia II - Nadolol - Mechanism of action |
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| | | | | | | Mechanism of action: Encyclopedia II - Ibuprofen - Mechanism of action Ibuprofen is an NSAID which is believed to work through inhibition of cyclooxygenase (COX); thus inhibiting prostaglandin synthesis. As with other NSAIDs, ibuprofen inhibits platelet aggregation, but is not used therapeutically for this action since it is a minor and reversible effect.
Main article: Non-steroidal anti-inflammatory drug
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See also:Ibuprofen, Ibuprofen - Clinical use, Ibuprofen - Indications, Ibuprofen - Off-Label and investigational use, Ibuprofen - Ibuprofen lysine, Ibuprofen - Mechanism of action, Ibuprofen - Adverse effects, Ibuprofen - Reported ADRs, Ibuprofen - Photosensitivity, Ibuprofen - Cardiovascular risk, Ibuprofen - Stereochemistry, Ibuprofen - Human toxicology, Ibuprofen - Availability Read more here: » Ibuprofen: Encyclopedia II - Ibuprofen - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Chlorpromazine - Mechanism of action
Chlorpromazine - Central.
Chlorpromazine acts as an antagonist (blocking agent) on different postsysnaptic receptors -on dopaminergic-receptors (subtypes D1, D2, D3 and D4 - different antipsychotic properties on productive and unproductive symptoms), on serotonergic-receptors (5-HT1 and 5-HT2, with anxiolytic, antidepressive and antiaggressive properties as well as an attenuation of extrapypramidal side-effects, but also leading to weight gain, fall in blood pressure, sedation and ejaculation difficulties), on hi ...
See also:Chlorpromazine, Chlorpromazine - Chemistry, Chlorpromazine - Mechanism of action, Chlorpromazine - Central, Chlorpromazine - Peripheral, Chlorpromazine - History, Chlorpromazine - Side effects, Chlorpromazine - Interactions, Chlorpromazine - Uses, Chlorpromazine - Common uses, Chlorpromazine - Off-label and controversial uses, Chlorpromazine - Veterinary uses, Chlorpromazine - Dosage, Chlorpromazine - Necessary examinations and laboratory checks during treatment, Chlorpromazine - Discontinuation of treatment, Chlorpromazine - Sources Read more here: » Chlorpromazine: Encyclopedia II - Chlorpromazine - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Sildenafil - Mechanism of action Part of the physiological process of erection involves the release of nitric oxide (NO) in the corpus cavernosum. This then activates the enzyme guanylate cyclase which results in increased levels of cyclic guanosine monophosphate (cGMP), leading to smooth muscle relaxation in the corpus cavernosum, resulting in increased inflow of blood and an erection.
Sildenafil is a potent and selective inhibitor of cGMP specific phosphodiesterase type 5 (PDE5) which is responsible for degradation of cGMP in the corpus cavernosum. This means that, ...
See also:Sildenafil, Sildenafil - History, Sildenafil - Mechanism of action, Sildenafil - Dosage and price, Sildenafil - Contraindications and side effects, Sildenafil - Uses outside of erectile dysfunction, Sildenafil - Pulmonary hypertension, Sildenafil - Raynaud's phenomenon Read more here: » Sildenafil: Encyclopedia II - Sildenafil - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Indomethacin - Mechanism of action Indomethacin is a nonselective inhibitor of cyclooxygenase (COX) 1 and 2, enzymes that participate in prostaglandin synthesis from arachidonic acid. Prostaglandins are hormone-like molecules normally found in the body, where they have a wide variety of effects, some of which lead to pain, fever, and inflammation.
Prostaglandins also cause uterine contractions in pregnant women. Indomethacin is an effective tocolytic agent, able to delay premature labor by reducing uterine contractions through inhibition of PG synthesis in the uterus a ...
See also:Indomethacin, Indomethacin - Chemical properties, Indomethacin - Indications, Indomethacin - Contraindications, Indomethacin - Mechanism of action, Indomethacin - Adverse effects, Indomethacin - Necessary Examinations during Longterm Treatment, Indomethacin - Animal Toxicity and Human Overdose, Indomethacin - Usual Dosage Forms, Indomethacin - History Read more here: » Indomethacin: Encyclopedia II - Indomethacin - Mechanism of action |
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| | | | | | Mechanism of action: Encyclopedia II - Capsaicin - Mechanism of action The burning and painful sensations associated with capsaicin result from capsaicin's chemical interaction with sensory neurons. Capsaicin, as a member of the vanilloid family, binds to a receptor called the vanilloid receptor subtype 1 (VR1). First cloned in 1997, VR1 is an ion channel-type receptor. VR1, which can also be stimulated with heat and physical abrasion, permits positively-charged ions (i.e. cations) to pass through the cell membrane and into the cell from outside when activated. The resulting "depolarization" of the neuron stimu ...
See also:Capsaicin, Capsaicin - Capsaicinoids, Capsaicin - Uses, Capsaicin - Food, Capsaicin - Medical, Capsaicin - Non-lethal force, Capsaicin - Pest deterrent, Capsaicin - Mechanism of action, Capsaicin - Capsaicin high Read more here: » Capsaicin: Encyclopedia II - Capsaicin - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Chlorpromazine - Mechanism of action
Chlorpromazine - Central.
Chlorpromazine acts as an antagonist (blocking agent) on different postsysnaptic receptors -on dopaminergic-receptors (subtypes D1, D2, D3 and D4 - different antipsychotic properties on productive and unproductive symptoms), on serotonergic-receptors (5-HT1 and 5-HT2, with anxiolytic, antidepressive and antiaggressive properties as well as an attenuation of extrapypramidal side-effects, but also leading to weight gain, fall in blood pressure, sedation and ejaculation difficulties), on hi ...
See also:Chlorpromazine, Chlorpromazine - Chemistry, Chlorpromazine - Mechanism of action, Chlorpromazine - Central, Chlorpromazine - Peripheral, Chlorpromazine - History, Chlorpromazine - Side effects, Chlorpromazine - Interactions, Chlorpromazine - Cancer risk carcinogenecity, Chlorpromazine - Uses, Chlorpromazine - Common uses, Chlorpromazine - Off-label and controversial uses, Chlorpromazine - Veterinary uses, Chlorpromazine - Dosage, Chlorpromazine - Necessary examinations and laboratory checks during treatment, Chlorpromazine - Discontinuation of treatment, Chlorpromazine - Sources Read more here: » Chlorpromazine: Encyclopedia II - Chlorpromazine - Mechanism of action |
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| | | | | Mechanism of action: Encyclopedia II - Amiodarone - Mechanism of action Amiodarone is categorized as a class III antiarrhythmic agent, and prolongs phase 3 of the cardiac action potential. It has numerous other effects however, including actions that are similar to those of antiarrhythmic classes Ia, II, and IV.
Amiodarone shows beta blocker-like and calcium channel blocker-like actions on the SA and AV nodes, increases the refractory period via sodium- and potassium-channel effects, and slows intra-cardiac conduction of the car ...
See also:Amiodarone, Amiodarone - History, Amiodarone - Dosing, Amiodarone - Mechanism of action, Amiodarone - Indications for use, Amiodarone - Ventricular fibrillation, Amiodarone - Ventricular tachycardia, Amiodarone - Atrial fibrillation, Amiodarone - Contraindications, Amiodarone - Metabolism, Amiodarone - Interactions with other drugs, Amiodarone - Excretion, Amiodarone - Side effects, Amiodarone - Thyroid, Amiodarone - Eye, Amiodarone - Gastrointestinal system, Amiodarone - Skin, Amiodarone - Lung, Amiodarone - Related topics Read more here: » Amiodarone: Encyclopedia II - Amiodarone - Mechanism of action |
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