Herbicide: Encyclopedia II - Herbicide - Classification of herbicides

Herbicide - Classification of herbicides

Herbicides can be grouped by activity, use, chemical family, mode of action, or type of vegetation controlled.

By activity:

• Contact herbicides destroy only the plant tissue in contact with the chemical. Generally, these are the fastest acting herbicides. They are less effective on perennial plants, which are able to regrow from roots or tubers.

• Systemic herbicides are translocated through the plant, either from foliar application down to the roots, or from soil application up to the leaves. They can destroy a greater amount of plant tissue than contact herbicides.

By use:

• Soil-applied herbicides are applied to the soil and are taken up by the roots of the target plant.

• Pre-emergent herbicides are applied to the soil before the crop emerges and prevent germination or early growth of weed seeds.

• Post-emergent herbicides are applied after the crop has emerged.

Their classification by mechanism of action (MOA) indicates the first enzyme, protein, or biochemical step affected in the plant following application. The main mechanisms of action are:

• ACCase inhibitors are compounds that kill grasses. Acetyl coenzyme A carboxylase (ACCase) is part of the first step of lipid synthesis. Thus, ACCase inhibitors affect cell membrane production in the meristems of the grass plant. The ACCases of grasses are sensitive to these herbicides, whereas the ACCases of dicot plants are not.

• ALS inhibitors: the acetolactate synthase (ALS) enzyme (also known as acetohydroxyacid synthase, or AHAS) is the first step in the synthesis of the branched-chain amino acids valine, leucine, and isoleucine. These herbicides slowly starve affected plants of these amino acids which eventually leads to inhibition of DNA synthesis. They affect grasses and dicots alike. The ALS inhibitor family includes sulfonylureas (SUs), imidazolinones (IMIs), triazolopyrimidines (TPs), pyrimidinyl oxybenzoates (POBs), and sulfonylamino carbonyl triazolinones (SCTs).

• EPSPS inhibitors: The enolpyruvylshikimate 3-phosphate synthase enzyme EPSPS is used in the synthesis of the amino acids tryptophan, phenylalanine and tyrosine. They affect grasses and dicots alike. Glyphosate (Roundup) is a systemic EPSPS inhibitor but inactivated by soil contact.

• Synthetic auxin inaugurated the era of organic herbicides. They were discovered in the 1940s after a long study of the plant growth regulator auxin. Synthetic auxins mimic this plant hormone. They have several points of action on the cell membrane, and are effective in the control of dicot plants. 2,4-D is a synthetic auxin herbicide.

• Photosystem II inhibitors reduce electron flow from water to NADPH2+ at the photochemical step in photosynthesis. They bind to the Qb site on the D2 protein, and prevent quinone from binding to this site. Therefore, this group of compounds cause electrons to accumulate on chlorophyll molecules. As a consequence, oxidation reactions in excess of those normally tolerated by the cell occur, and the plant dies. The triazine herbicides (including atrazine) are PSII inhibitors.

Adapted from the Wikipedia article "Classification of herbicides", under the G.N U Free Docmentation License. Please also see http://en.wikipedia.org/wiki