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Aflatoxin - Pathology | | Aflatoxin - Pathology: Encyclopedia II - Aflatoxin - Pathology | | High-level aflatoxin exposure produces an acute necrosis, cirrhosis, and carcinoma of the liver exhibited by hemorrhage, acute liver damage, edema, alteration in digestion, and absorption and/or metabolism of nutrients.
No animal species is immune to the acute toxic effects of aflatoxins including humans; however, humans have an extraordinarily high tolerance for aflatoxin exposure and rarely succumb to acute aflatoxicosis.
Chronic, subclinical exposure does not lead to as dramatic of symptoms as acute aflatoxicosis. Children, ...
See also:Aflatoxin, Aflatoxin - Contamination Conditions, Aflatoxin - Pathology, Aflatoxin - Detection of Aflatoxin in Humans, Aflatoxin - Major types of Aflatoxins and its metabolite, Aflatoxin - Interaction of Aflatoxin with Hepatitis B Virus | | | Aflatoxin, Aflatoxin - Contamination Conditions, Aflatoxin - Detection of Aflatoxin in Humans, Aflatoxin - Interaction of Aflatoxin with Hepatitis B Virus, Aflatoxin - Major types of Aflatoxins and its metabolite, Aflatoxin - Pathology, Synthetic aflatoxins in Aflatoxin total synthesis | | |
| | | Aflatoxin: Encyclopedia II - Aflatoxin - Pathology
Aflatoxin - Pathology
High-level aflatoxin exposure produces an acute necrosis, cirrhosis, and carcinoma of the liver exhibited by hemorrhage, acute liver damage, edema, alteration in digestion, and absorption and/or metabolism of nutrients.
No animal species is immune to the acute toxic effects of aflatoxins including humans; however, humans have an extraordinarily high tolerance for aflatoxin exposure and rarely succumb to acute aflatoxicosis.
Chronic, subclinical exposure does not lead to as dramatic of symptoms as acute aflatoxicosis. Children, however, are particularly affected by aflatoxin exposure which leads to stunted growth and delayed development. Chronic exposure also leads to a high risk of developing liver cancer, as the metabolite Aflatoxin M1 can intercalate into DNA and alkylate the bases through its epoxide moiety.
Other related archives2003 invasion of Iraq, Aflatoxin total synthesis, Aspergillus, Aspergillus flavus, Aspergillus parasiticus, China, DNA, Food and Drug Administration, Hepatitis B, Iraq, Saddam Hussein, Salman Pak facility, UNSCOM, United States, albuminadduct, alkylate, almond, black pepper, carcinogenic, carcinoma of the liver, cereals, chile peppers, cirrhosis, coconut, coriander, cotton, edema, epoxide, ginger, guanine, half-life, hemorrhage, hepatocellular carcinoma, intercalate, liver, maize, moiety, mycotoxins, necrosis, ng, nuts, oilseeds, oncogenes, p53, peanut, pearl millet, pistachio, rice, sorghum, soybean, spices, sunflower, turmeric, vaccination, walnut, western Africa, wheat
 Adapted from the Wikipedia article "Pathology", under the G.N U Free Docmentation License. Please also see http://en.wikipedia.org/wiki |
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